External inflammatory root resorption can be further categorized into cervical resorption with or without a vital pulp (invasive cervical root resorption) and external apical root resorption. Other variations of resorption include combined internal and external resorption and transient apical breakdown.

Resorption initiation. The external surface of the root is surrounded by a protective layer called precementum. Damage to this layer allows resorptive cells (odontoclasts) to penetrate the root. The portal of entrance into the root can range in size from pinpoint to a much larger defect.

External inflammatory root resorptions are pathologies that are difficult to treat. The treatment aims to eliminate the cause or stimulating factors of clastic cells such as infection and/or pressure. The size and location of the lesions are limiting factors for the treatment and maintenance of the affected tooth. They can be classified as internal, when root canal is involved and external, when periodontal ligament is involved.

2021-04-08 · External resorption of a root (ERR) is a more common phenomenon. Along with being more common, most cases of external resorption are mild and seem to be self limiting. That being said, 10% of patients who present with external resorption do show concerning amounts of progression 1 . Internal resorption is the result of an inflamed pulp and the clastic precursor cells recruiting through the blood vessels. Treatment of internal resorption is quite predictable as it is easy to control the process of internal root resorption via severing the blood supply to the resorbing tissues with conventional root canal therapy.

Root resorption can be divided into two main categories: internal root resorption and external root resorption. Internal root resorption is a pathology that can lead to tooth destruction in the short term and must consequently be stopped as soon as possible by applying adequate canal treatment. Until now, despite many research studies, very

External root resorptions are processes in which the loss of cementoblasts occurs causing the mineralized surfaces to be susceptible to the action of clastic cells. (2) There are two different kinds of resorption — external and internal. External resorption is resorption initiated in the periodontium and initially affecting the external surfaces of a tooth — may be further classified as surface, inflammatory, or replacement, or by location as cervical, lateral, or apical; may or may not invade the dental pulp space. The outline of original root canal is not distorted and can be differentiated from external root resorption by changing radiographic angualtions.

2021-01-30

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1,2 Resorption is seen on the wall of the root canal (internal resorption) and on the external surface of the root (external resorption or cervical resorption) and it may be transient or progressive. 3 In the rest of this article, only intraradicular forms of internal root resorption will be discussed and and it will be simply referred to as internal resorption. Etiology For internal root resorption to occur, the outermost protective odontoblast layer and the predentin of the canal wall must be damaged, resulting in exposure of the underlying mineralized dentin to odontoclasts. 2018-01-17 · Unlike external resorption, this process is arrested once the pulpectomy is done or remaining pulp becomes completely non-vital. In a study conducted by Thoma et al.,13 the authors reported internal root resorption in only one tooth out of the 1000 teeth studied It has been shown that like the cells of the periodontal ligament which are responsible for external root resorption, the pulp External resorption is much more common than internal resorption, but is often misdiagnosed. Treatment for internal resorption includes root canal therapy, followed by a definitive restoration. Although root canal treatment will halt the resorptive process, if the defect is too large, the tooth may mechanically fracture and fail.
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The treatment of root resorption is basically root and Resorption initiation. The external surface of the root is surrounded by a protective layer called precementum. Damage to this layer allows resorptive cells (odontoclasts) to penetrate the root. The portal of entrance into the root can range in size from pinpoint to a much larger defect.

Internal root resorption is usually thought to be the result of long-standing chronic inflammation in your tooth’s nerve (technically known as the pulp) and is asymptomatic more often than not.
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of the cation exchange and lacunar resorption by osteoclasts. There are two types: external (as a result of tooth pathology) and internal (apparently initiated by

Note the loss of the canal space with the resorptive defect. Cleaning of the root canal space with internal resorptive defect in the maxillary central incisor Thus, root resorption in the permanent dentition is a pathologic event; if untreated, this might result in the premature loss of the affected teeth.Root resorption might be broadly classified into external or internal resorption by the location of the resorption in relation to the root surface (6,7). Root resorption might be broadly classified into external or internal resorption by This might be due, in part, to the relatively rare occur- the location of the resorption in relation to the root surface (6, 7). Internal root rence of this type of resorption and the lack of an in resorption … In the rest of this article, only intraradicular forms of internal root resorption will be discussed and and it will be simply referred to as internal resorption. Etiology For internal root resorption to occur, the outermost protective odontoblast layer and the predentin of the canal wall must be damaged, resulting in exposure of the underlying mineralized dentin to odontoclasts. Invasive Cervical Root Resorption, External Cervical Resorption, and External invasive cervical root resorption – Oh My! Lots of names but all the same biological process and same are synonyms. Cases with External cervical resorption or internal resorption that I am not treating but monitoring.